@kiranbatta.bsky.social
We are seeking a highly motivated PhD candidate to investigate how altered communication between leukaemic stem cells and the bone marrow niche drives leukemogenesis.
November 12, 2025 at 10:08 AM
We are seeking a highly motivated PhD candidate to investigate how altered communication between leukaemic stem cells and the bone marrow niche drives leukemogenesis.
Huge congratulations to Yu-Hung Wang, who has been honoured with the Best Outstanding Research Contribution Award from the FBMH Doctoral Academy at the University of Manchester! We are incredibly proud of Yu-Hung's dedication and hard work!
July 2, 2025 at 1:57 PM
Huge congratulations to Yu-Hung Wang, who has been honoured with the Best Outstanding Research Contribution Award from the FBMH Doctoral Academy at the University of Manchester! We are incredibly proud of Yu-Hung's dedication and hard work!
Resistin also induces myeloid-derived immune suppressive features in monocytes, leading to an expansion of regulatory T cells. @uom-dcs.bsky.social @mcrcnews.bsky.social
March 10, 2025 at 11:07 AM
Resistin also induces myeloid-derived immune suppressive features in monocytes, leading to an expansion of regulatory T cells. @uom-dcs.bsky.social @mcrcnews.bsky.social
www.biorxiv.org/content/10.1...
In this preprint, we showed that granulocytes from CMML patients are immature and produce high levels of an adipokine called Resistin. Resistin binds to monocytes and prevents their maturation, potentially contributing to the monocytosis phenotype in CMML.
In this preprint, we showed that granulocytes from CMML patients are immature and produce high levels of an adipokine called Resistin. Resistin binds to monocytes and prevents their maturation, potentially contributing to the monocytosis phenotype in CMML.
March 10, 2025 at 11:07 AM
www.biorxiv.org/content/10.1...
In this preprint, we showed that granulocytes from CMML patients are immature and produce high levels of an adipokine called Resistin. Resistin binds to monocytes and prevents their maturation, potentially contributing to the monocytosis phenotype in CMML.
In this preprint, we showed that granulocytes from CMML patients are immature and produce high levels of an adipokine called Resistin. Resistin binds to monocytes and prevents their maturation, potentially contributing to the monocytosis phenotype in CMML.