Mariona Graupera
@grauperalab.bsky.social
Vascular biologist, vascular malformations and genetic mosaicisims
Finally, thank you PTEN Research Foundation, for supporting our work
March 28, 2025 at 3:12 PM
Finally, thank you PTEN Research Foundation, for supporting our work
GrauperaLab
@MarionaGraupera
·
2min
This is a team effort with many involved, but I would especially thank @sandradcastillo.bsky.social the driving force of this work, and Eulàlia Baselga the clinician managing these patients
@MarionaGraupera
·
2min
This is a team effort with many involved, but I would especially thank @sandradcastillo.bsky.social the driving force of this work, and Eulàlia Baselga the clinician managing these patients
March 28, 2025 at 3:12 PM
GrauperaLab
@MarionaGraupera
·
2min
This is a team effort with many involved, but I would especially thank @sandradcastillo.bsky.social the driving force of this work, and Eulàlia Baselga the clinician managing these patients
@MarionaGraupera
·
2min
This is a team effort with many involved, but I would especially thank @sandradcastillo.bsky.social the driving force of this work, and Eulàlia Baselga the clinician managing these patients
By demonstrating that the differentiation state and the fate of endothelial cells dictate the cellular response to PIK3CA mutations, we provide a novel paradigm for understanding the mechanisms dictating tissue-specific overgrowth in PIK3CA-related congenital disorders
February 26, 2025 at 12:19 PM
By demonstrating that the differentiation state and the fate of endothelial cells dictate the cellular response to PIK3CA mutations, we provide a novel paradigm for understanding the mechanisms dictating tissue-specific overgrowth in PIK3CA-related congenital disorders
Our findings expose the (i) generation of large clones in veins and capillaries causing vascular malformations, (ii) silent presence in arterial cells without pathological manifestations; and (iii) rewire of cell fate in arterial precursors
February 26, 2025 at 12:19 PM
Our findings expose the (i) generation of large clones in veins and capillaries causing vascular malformations, (ii) silent presence in arterial cells without pathological manifestations; and (iii) rewire of cell fate in arterial precursors